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Muscular Dystrophy

A fitness pill. Credit: The Science website via DALEE

New 'workout pill' can cause fitness benefits without exercise

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Researchers report new compounds that appear to be able to mimic the physical drive of exercise—at least in rodent cells. This discovery could lead to a new way to treat muscular dystrophy and other medical conditions in humans, including heart failure and neurodegenerative diseases
Left: cells with a mutant FUS protein that causes accumulations (in green) and leads to ALS disease. Right: the cells with the protein/chaferon (in pink) that manages to prevent the accumulations

fight degenerative diseases

The proteins and mechanisms required to fight the protein accumulations that lead to neurodegenerative diseases have been identified
Microglial cells that were "ripened" in the laboratory from stem cells derived from ALS patients (in green), the cell nuclei - in blue. Photographed using a confocal microscope

The good mutation

Weizmann Institute of Science scientists have discovered a new pathway that may protect nerve cells in the brain and slow down the development of degenerative brain disease
Neurons of a mouse embryo grown in a Petri dish. The axons, the extensions of the nerve cells, are the green extensions in the picture. Source: NIH.

A little less, a little more

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Treatment of an ALS patient. Source: Pison Technology website, one of the companies that won the award.

Peres Chaim Association awarded a grant of 600 thousand dollars for the development of innovative communication systems for ALS patients

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DNA replication using RNA. Illustration: shutterstock

Researchers from the Hebrew University have cracked a mechanism that will contribute to the understanding of muscular dystrophy

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