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Keep an eye on the pigs!

Would tracking animals have made it possible to predict the swine flu?

Swine Flu
Swine Flu

By Christine Soares

In January 2009, a passenger plane had to make an emergency landing in the waters of the Hudson River in New York. In less than 24 hours, video footage from several surveillance cameras installed in the area was published, documenting the event from different angles. Nowadays, with many surveillance systems deployed everywhere, people have become accustomed to assuming that someone or something is always watching them, ready to spot problems the moment they occur. Despite this, in March and April 2009, a new strain of influenza A, H1N1, managed to pass from pigs to humans, and as of the end of July, health and agricultural officials are still trying to understand where it came from.

The appearance of the H1N1 flu strain proved both the effectiveness of the existing systems for monitoring flu outbreaks among humans, and the theory that pigs may serve as a transit point for viruses from animals to humans. But she also emphasized how disappointing our ability to find out where such viruses come from, to understand how they develop in animals and to predict if they will infect humans, an ability that would have allowed us to prevent an epidemic or at least warn of it.

Despite years of awareness of the issue and budgets allocated to influenza research, health officials have not yet been able to find an effective way to warn against new animal pathogens that may be transmitted to humans. For example, Jorgen A. Richt and his colleagues at the US Department of Agriculture's National Animal Disease Center in Ames, Iowa, identified a new strain of influenza A, H2007N2, in pigs in 3, which they believed could become a pandemic. But "there was no one to tell about it," according to Richt. "So we asked ourselves: 'What do we do with this information?' It's nobody's concern, because there were no laws or regulations on the matter." Richt and his colleagues therefore published their hypothesis in a scientific journal, and concluded that "it would be wise to establish a surveillance system for diseases in pigs and workers exposed to them."

When it comes to diseases, surveillance should include, at the very least, doctors and laboratories that will report any pathogen they identify. For example, in the USA, you can report every case of influenza in humans to the American Centers for Disease Control and Prevention (CDC), which monitors cases of the disease and its spread. But laboratory tests done of the free will of the doctors, both in humans and animals, reflect only a small proportion of the cases that reached a doctor's visit. The obligation to perform systematic tests and report their results currently only applies to pig diseases that can cause severe economic damage, such as swine fever and nipa disease.

According to Richt, who currently works at the University of Kansas, veterinary laboratories for disease detection can play an important role in more active monitoring of animal diseases, if only they carry out a comprehensive test, of all possible disease agents, in every sample they receive, regardless of the reason the sample was sent.

"We need a better network for monitoring animal populations, so that we can detect infectious diseases in their initial stages with the innovative technology of the 21st century," says Richt. The large government laboratories in the states where the largest pig populations in the US are found, such as Iowa and North Carolina, already have the technology needed to track a range of pig diseases, explains Richt. Smaller laboratories can detect certain pathogens in pigs, cattle or poultry with the help of DNA arrays, thus providing a more comprehensive picture of the diseases that can be transmitted to humans, such as the new strain of influenza, in real time, while they are still in the stages of development in the animal population.

But let's not forget that discovering new flu strains in animals is one thing, and being able to determine whether such a particular strain might endanger humans is another. "I'm much more pessimistic about our ability to predict this," explains Jeffrey K. Taubenberger of the American Institute of Infectious Diseases and Allergy. In March 2009 he published an analysis of two substrains of the H1N1 family in pigs. One of them was the Eurasian strain that contributed DNA segments to the new strain that attacks humans today. The two strains share a common H1N1 progenitor, but they evolved independently in pig populations, and the tiny changes in the viral genes, which allow the virus to adapt to a new animal, were different in each substrain. Many scientists who have looked for consistent signs that some virus is about to become more contagious or virulent have failed to discover clear patterns.
That's why no one knows how to explain why the H5N1 bird flu virus, which infected about 400 people around the world, especially in Asia and Africa, has so far failed to fully adapt to humans. Scientists also have no idea where the flu virus that caused the 1918 pandemic originated, or where its distant descendant, the new strain of H1N1, is headed. After spreading around the world, it infected more than 130,000 people and claimed the lives of more than 800, as of the end of July 2009. The new virus may weaken in the coming months, or rather learn how to make the transition from person to person more efficient. In the fall of 2010 he may return to the Northern Hemisphere strong as a lion, or meek as a sheep.

Taubenberger, who was one of the US Army Pathology Institute researchers who in 1996 isolated for the first time from preserved tissue the historical H1N1 strain that caused the 1918 flu pandemic, explains that there is more hidden than visible in the basic biology and ecology of influenza viruses. In his opinion, monitoring an entire rural ecosystem: pigs, poultry, humans as well as dogs, cats, horses and other domestic and wild animals, could finally yield a deeper understanding of the way and reason for the development of influenza viruses.
Fortunately, the budgets allocated to pandemic defense have greatly improved the surveillance and response systems for human influenza. Richt points out that laboratories in the US identified the new flu very quickly in two children in Southern California and alerted the CDC, thus enabling a quick response by the US health system. Unfortunately, in the absence of closer monitoring of animals that may be the source of new influenza strains, monitoring humans will continue to be our first line of defense.

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5 תגובות

  1. This is true for Ariel, after this commenter failed to meet the minimum conditions for participating in the discussion and kept hijacking it (see various articles from last week). There was no choice but to limit it so that others could also participate in the discussion.

  2. Ron, do you really believe what you say??
    Since the 50s there have been almost no cases of polio, why? Because of improved hygiene? sewerage? Do you think living conditions have changed that much? Despite this, the disease disappeared.
    Where are the smallpox? The disease that destroyed the Native American civilizations, killing hundreds of millions of people throughout history, miraculously disappeared. So it was no wonder, Jenner invented an effective vaccine.

    It is clear that there are ineffective vaccines and I would not automatically run to take every vaccine, but cancel all vaccines with a sweeping hand?

  3. Father, please also let this comment remain.

    Regarding graphs on distribution and mortality from certain diseases before and after they started to vaccinate:
    What you claim is a myth spread with malicious intent.
    In reality - the epidemics subsided naturally due to the improvement in living conditions - sewage, clean food and water, hygiene, etc.
    We have a record of formal research from the
    Official Year Books of the Commonwealth of Australia

    Look at the graphs
    http://www.harvestdream.org/index.php?/archives/431-GRAPHICAL-EVIDENCE-SHOWS-VACCINES-DIDNT-SAVE-US.html

  4. The first commenter probably cited vanities, conspiracies and mistakes.
    For example: one protein of the current virus is similar to the 1918 virus, while another protein is not at all. Regarding the vaccine, I don't even know how to begin to address this tremendous and dangerous nonsense. Just look for graphs on prevalence and mortality from certain diseases before and after they started vaccinating.

    Abi, "I am limited to responding per article - I cannot respond to questions or comments" Is this really true?

  5. We know about this virus beyond any reasonable doubt what its origin is.

    The 2009 outbreak occurred near Baxter Laboratories outside Mexico City. 

    Please familiarize yourself with our virus, and all its piracy and genetic origins
    Information from Alexander S. Jones formerly of NIH
    National Institutes of Health
    One clear picture
    labvirus.files.wordpress.com/2009/09/39b02.png

    Is the virus engineered? Without a doubt
    It is a fact that Dr. Jeffrey Taubenberger
    began working in the mid-90's
    on reverse engineering the deadly 1918
    virus that killed untold millions of people.
    -
    It is a fact that he assembled his
    "team" at the US Army Institute of
    Pathology in 1996 and officially began
    work on the "project" in 1997.
    -
    It is a fact that in 1997, Taubenberger
    published a paper in the journal "Science"
    identifying that the 1918 killer virus
    was a "triple reassortant novel" virus
    containing genetic segments of
    Human, Swine, and Avian origin.
    Taubenberger completed his "project" in 2003
    , then begins working for the NIH
    in the area of ​​vaccine development.
    -
    In 2005, a Wisconsin boy was diagnosed
    with the same triple virus
    now declared a pandemic by the WHO
    in 2009, SHORTLY AFTER RECEIVING A FLU SHOT.
    -
    It is also a fact that the world's largest
    multinational pharmaceutical corporation, Novartis,
    applied for a patent for a vaccine
    designed to address a "reverse-engineered novel
    triple reassortant pandemic virus” on November 4, 2005
    -
    - just a week before the Wisconsin boy
    receives his flu vaccine - and a month before
    the boy develops the first case of "triple" virus.
    -
    This patent was granted in February of 2009
    , then a very short month later, the same "triple-virus"
    is publicized worldwide as a terribly deadly Mexican outbreak. Within days, Novartis receives billions of
    dollars in vaccine contracts from panicked nations.
    -
    Fact:: the "novel swine flu" has
    an extremely low mortality rate
    compared with "seasonal flu".
    who.int/csr/don/2009_05_26/en/index.html
    In fact, the CDC and the WHO have
    quit listing the cases and the fatalities,
    quite possibly because of this fact.
    -
    They may be concerned that people will begin to ask:
    "This doesn't appear to be that deadly,
    why all the hysteria calling for a headlong
    rush to implement mass vaccination in our schools
    and in pregnant women?” The same people would
    likely then ask in the next breath:
    "Could it be possible that the vaccine
    can likely be many times more
    dangerous than the disease itself??”
    -
    Fact: I have shown that the CDC and the WHO
    have mistaken America as to the history and origins
    of the "Triple Virus", so why should we believe
    their projections about the coming waves of death
    unless vaccines are pushed?
    -
    An analysis of the "swine flu" genome sequence
    by Alexander S Jones NIH Whistleblower
    indicates that only 5% of the "Novel Swine"
    pandemic influenza A RNA sequences
    (swine flu combined with avian flu viruses
    identified in the "pandemic" 2009 virus
    share no known homology in any public databases
    in addition to the avian/swine hybrid
    nature of both these critical genes,
    -
    and so a laboratory origin for this virus
    is the only logical conclusion.
    -
    95% if the sequences are readily available
    to microbiologists working in
    Department of Defense weapons labs
    – i.e. Ft. Detrick, and can be "spliced"
    together to form a near-perfect match
    to a previously identified viral pathogen
    -
    The source article
    labvirus.wordpress.com/2009/08/01
    evidence-of-actual-origins-of-h1n1-swine-flu/

    ----

    The story is very simple
    This is a bridge virus
    The spreaders of the virus are doing everything to make this virus violent
    It lacks only two amino acids to resemble the 1918 flu
    The vaccine, some of which contain parts of the DNA of the deadly bird flu and cotyl of toxins such as aluminum and mercury, and the weakening of the human immune system actually creates a hotbed and activates the growth of an aggressive and deadly virus.
    You create a virus and cover your tracks.
    Conversation from April 2009
    With Dr. Bill Diggle
    Minute 8: 44
    http://www.youtube.com/watch?v=rk_U5_dNyqo

    I am limited to one response per article by the site administrator - I cannot respond to questions or comments.

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