Would tracking animals have made it possible to predict the swine flu?
By Christine Soares
In January 2009, a passenger plane had to make an emergency landing in the waters of the Hudson River in New York. In less than 24 hours, video footage from several surveillance cameras installed in the area was published, documenting the event from different angles. Nowadays, with many surveillance systems deployed everywhere, people have become accustomed to assuming that someone or something is always watching them, ready to spot problems the moment they occur. Despite this, in March and April 2009, a new strain of influenza A, H1N1, managed to pass from pigs to humans, and as of the end of July, health and agricultural officials are still trying to understand where it came from.
The appearance of the H1N1 flu strain proved both the effectiveness of the existing systems for monitoring flu outbreaks among humans, and the theory that pigs may serve as a transit point for viruses from animals to humans. But she also emphasized how disappointing our ability to find out where such viruses come from, to understand how they develop in animals and to predict if they will infect humans, an ability that would have allowed us to prevent an epidemic or at least warn of it.
Despite years of awareness of the issue and budgets allocated to influenza research, health officials have not yet been able to find an effective way to warn against new animal pathogens that may be transmitted to humans. For example, Jorgen A. Richt and his colleagues at the US Department of Agriculture's National Animal Disease Center in Ames, Iowa, identified a new strain of influenza A, H2007N2, in pigs in 3, which they believed could become a pandemic. But "there was no one to tell about it," according to Richt. "So we asked ourselves: 'What do we do with this information?' It's nobody's business, because there were no laws or regulations on the matter." Richt and his colleagues therefore published their hypothesis in a scientific journal, concluding that "it would be prudent to establish a surveillance system for diseases in pigs and workers exposed to them."
When it comes to diseases, surveillance should include, at the very least, doctors and laboratories that will report any pathogen they identify. For example, in the US, every case of influenza in humans can be reported to the US Centers for Disease Control and Prevention (CDC), which tracks cases of the disease and its spread. But laboratory tests done voluntarily by doctors, both in humans and animals, reflect only a small proportion of The cases that have reached a doctor's visit. The obligation to perform systematic tests and report their results currently applies only to pig diseases that can cause severe economic damage, such as swine fever and Nipah disease.
According to Richt, who currently works at the University of Kansas, veterinary laboratories for disease detection can play an important role in more active monitoring of animal diseases, if only they carry out a comprehensive test, of all possible disease agents, in every sample they receive, regardless of the reason the sample was sent.
"We need a better network for monitoring animal populations, so that we can detect infectious diseases in the initial stages with the help of the innovative technology of the 21st century," says Richt. The large government laboratories in states with the largest pig populations in the US, such as Iowa and North Carolina, already have the technology needed to monitor a spectrum of swine diseases, Richt explains. Smaller laboratories can detect certain pathogens in pigs, cattle or poultry using DNA arrays. , and thus provide a more comprehensive picture of the diseases that can be transmitted to humans, such as the new strain of influenza, in real time, while they are still in the stages of development in the animal population.
But let's not forget that discovering new flu strains in animals is one thing, and being able to determine whether such a particular strain might endanger humans is another. "I'm much more pessimistic about our ability to predict this," explains Jeffrey K. Taubenberger of the US Institute of Infectious Diseases and Allergy. In March 2009 he published an analysis of two substrains of the H1N1 family in pigs. One of them was the Eurasian strain that contributed DNA segments to the new strain that attacks humans today. The two strains shared a common H1N1 parent, but they evolved independently in pig populations, and the tiny changes in the viral genes, which allow the virus to adapt to a new animal, were different in each sub -Strain Many scientists who have looked for consistent signs that a virus is about to become more contagious or virulent have failed to discover clear patterns.
That's why no one knows how to explain why the H5N1 bird flu virus, which infected about 400 people around the world, especially in Asia and Africa, has so far failed to fully adapt to humans. Scientists also have no idea where the flu virus that caused the 1918 pandemic originated, or where its distant descendant, the new strain of H1N1, is headed. After spreading around the world, it infected more than 130,000 people and claimed the lives of more than 800, as of the end of July 2009. The new virus may weaken in the coming months, or rather learn how to make the transition from person to person more efficient. In the fall of 2010 he may return to the Northern Hemisphere strong as a lion, or meek as a sheep.
Taubenberger, who was one of the US Army Pathology Institute researchers who in 1996 isolated for the first time from preserved tissue the historical H1N1 strain that caused the 1918 flu pandemic, explains that there is more hidden than visible in the basic biology and ecology of influenza viruses. In his opinion, monitoring an entire rural ecosystem: pigs, poultry, humans as well as dogs, cats, horses and other domestic and wild animals, could finally yield a deeper understanding of the way and reason for the development of influenza viruses.
Fortunately, the budgets allocated to pandemic defense have greatly improved the surveillance and response systems for human influenza. Richt points out that laboratories in the US identified the new flu very quickly in two children in Southern California and alerted the CDC, thus enabling a quick response by the US health system. Unfortunately, in the absence of closer monitoring of animals that may be the source of new influenza strains, monitoring humans will continue to be our first line of defense.
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This is true for Ariel, after this commenter failed to meet the minimum conditions for participating in the discussion and kept hijacking it (see various articles from last week). There was no choice but to limit it so that others could also participate in the discussion.
Ron, do you really believe what you say??
Since the 50s there have been almost no cases of polio, why? Because of improved hygiene? sewerage? Do you think living conditions have changed that much? Despite this, the disease disappeared.
Where are the smallpox? The disease that destroyed the Native American civilizations, killing hundreds of millions of people throughout history, miraculously disappeared. So it was no wonder, Jenner invented an effective vaccine.
It is clear that there are ineffective vaccines and I would not automatically run to take every vaccine, but cancel all vaccines with a sweeping hand?
Father, please also let this comment remain.
Regarding graphs on distribution and mortality from certain diseases before and after they started to vaccinate:
What you claim is a myth spread with malicious intent.
In reality - the epidemics subsided naturally due to the improvement in living conditions - sewage, clean food and water, hygiene, etc.
We have a record of formal research from the
Official Year Books of the Commonwealth of Australia
Look at the graphs
http://www.harvestdream.org/index.php?/archives/431-GRAPHICAL-EVIDENCE-SHOWS-VACCINES-DIDNT-SAVE-US.html
The first commenter probably cited vanities, conspiracies and mistakes.
For example: one protein of the current virus is similar to the 1918 virus, while another protein is not at all. Regarding the vaccine, I don't even know how to begin to address this tremendous and dangerous nonsense. Just look for graphs on prevalence and mortality from certain diseases before and after they started vaccinating.
Abi, "I am limited to responding per article - I cannot respond to questions or comments" Is this really true?
We know about this virus beyond any reasonable doubt what its origin is.
The 2009 outbreak occurred near Baxter Laboratories outside Mexico City.
Please familiarize yourself with our virus, and all its piracy and genetic origins
Information from Alexander S. Jones formerly of NIH
National Institutes of Health
One clear picture
labvirus.files.wordpress.com/2009/09/39b02.png
—
Is the virus engineered? Without a doubt
It is a fact that Dr. Jeffrey Taubenberger
began working in the mid-90's
on reverse engineering the deadly 1918
virus that killed untold millions of people.
-
It is a fact that he assembled his
"team" at the US Army Institute of
Pathology in 1996 and officially began
work on the "project" in 1997.
-
It is a fact that in 1997, Taubenberger
published a paper in the journal "Science"
identifying that the 1918 killer virus
was a "triple reassortant novel" virus
containing genetic segments of
Human, Swine, and Avian origin.
Taubenberger completed his "project" in 2003
, then begins working for the NIH
in the area of vaccine development.
-
In 2005, a Wisconsin boy was diagnosed
with the same triple virus
now declared a pandemic by the WHO
in 2009, SHORTLY AFTER RECEIVING A FLU SHOT.
-
It is also a fact that the world's largest
multinational pharmaceutical corporation, Novartis,
applied for a patent for a vaccine
designed to address a "reverse-engineered novel
triple reassortant pandemic virus” on November 4, 2005
-
- just a week before the Wisconsin boy
receives his flu vaccine - and a month before
the boy develops the first case of "triple" virus.
-
This patent was granted in February of 2009
, then a very short month later, the same "triple-virus"
is publicized worldwide as a terribly deadly Mexican outbreak. Within days, Novartis receives billions of
dollars in vaccine contracts from panicked nations.
-
Fact:: the "novel swine flu" has
an extremely low mortality rate
compared with "seasonal flu".
who.int/csr/don/2009_05_26/en/index.html
In fact, the CDC and the WHO have
quit listing the cases and the fatalities,
quite possibly because of this fact.
-
They may be concerned that people will begin to ask:
"This doesn't appear to be that deadly,
why all the hysteria calling for a headlong
rush to implement mass vaccination in our schools
and in pregnant women?” The same people would
likely then ask in the next breath:
"Could it be possible that the vaccine
can likely be many times more
dangerous than the disease itself??”
-
Fact: I have shown that the CDC and the WHO
have mistaken America as to the history and origins
of the "Triple Virus", so why should we believe
their projections about the coming waves of death
unless vaccines are pushed?
-
An analysis of the "swine flu" genome sequence
by Alexander S Jones NIH Whistleblower
indicates that only 5% of the "Novel Swine"
pandemic influenza A RNA sequences
(swine flu combined with avian flu viruses
identified in the "pandemic" 2009 virus
share no known homology in any public databases
in addition to the avian/swine hybrid
nature of both these critical genes,
-
and so a laboratory origin for this virus
is the only logical conclusion.
-
95% if the sequences are readily available
to microbiologists working in
Department of Defense weapons labs
– i.e. Ft. Detrick, and can be "spliced"
together to form a near-perfect match
to a previously identified viral pathogen
-
The source article
labvirus.wordpress.com/2009/08/01
evidence-of-actual-origins-of-h1n1-swine-flu/
Sigrún has taught at the Iceland University of the Arts as a part-time lecturer since XNUMX and was Dean of the Department of Fine Art from XNUMX-XNUMX. In XNUMX–XNUMX she held a research position at Reykjavík Art Museum focusing on the role of women in Icelandic art. She studied fine art at the Icelandic College of Arts and Crafts and at Pratt Institute, New York, and holds BA and MA degrees in art history and philosophy from the University of Iceland. Sigrún lives and works in Iceland.
The story is very simple
This is a bridge virus
The spreaders of the virus are doing everything to make this virus violent
It lacks only two amino acids to resemble the 1918 flu
The vaccine, some of which contain parts of the DNA of the deadly bird flu and cotyl of toxins such as aluminum and mercury, and the weakening of the human immune system actually creates a hotbed and activates the growth of an aggressive and deadly virus.
You create a virus and cover your tracks.
Conversation from April 2009
With Dr. Bill Diggle
Minute 8: 44
http://www.youtube.com/watch?v=rk_U5_dNyqo
I am limited to one response per article by the site administrator - I cannot respond to questions or comments.